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Most of these proteins act as chemotactic factors at the site of inflammation, promoting the initial recruitment of satellite cells, neutrophils, monocytes, and, later, lymphocytes for tissue repair [52, 62,63,64,65]

Physiologically, c-Met is responsible for the cell-scattering phenotype, as first demonstrated with MDCK cells treated with HGF

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If a simple tripeptide could shift old tissue behavior toward a younger pattern, it suggested a broader role in tissue maintenance and repair that might operate across multiple organ systems